Neuroscience News

New Alzheimer's biomarkers focus on whether Alzheimer's has actually begun to affect a person's thinking and memory, rather than just amyloid levels.

This week on the podcast, Stanford psychiatry professor Erin Gibson joins us again to share the latest findings on sleep, myelin, and neurodegenerative disease.

New findings about the sugary armor on the brain’s frontline cells could shed light on cognitive decline and diseases like Alzheimer’s—and open new avenues f

How does the brain wire itself for learning? What molecular mechanisms protect neural circuits during aging? These are just some of the research projects by the 2025 Neurosciences Postdoctoral Scholars.

Research supported by a Knight Initiative Catalyst Grant explores whether targeting pathways related to the brain’s “chill-out” system could restore youthful resilience and improve cognitive function.

Researchers at Stanford’s Knight Initiative for Brain Resilience merge spatial transcriptomics and AI to uncover how local cellular interactions drive brain aging and resilience

New research shows aging is not a uniform process. Parts of our bodies start aging earlier than others, right down to our organs and cells.

Fat-filled microglia may play a role in the damage caused by Alzheimer’s, especially for those who carry two copies a particular gene.

With Knight Initiative support, Stanford researchers uncover a biochemical “off-ramp” in ketosis, rewriting our understanding of how ketosis influences metabolism.

The Knight Initiative for Brain Resilience supports 14 high-risk, high-reward projects to advance brain health and resilience research, fostering bold research across various scientific disciplines.

This week on From Our Neurons to Yours, we talk with Stanford neurologist Mike Greicius about his critique of new amyloid-clearing Alzheimer's drugs, and his optimism for the next wave of therapies currently in development.

Study in mice, supported by the Knight Initiative, suggests that an experimental cancer drug boosted brain metabolism, restored memory, and improved Alzheimer's symptoms. This novel approach may offer a new way to treat Alzheimer's beyond targeting amyloid plaques.

The APOE4 genetic variant, carried by about 20% of people, significantly raises Alzheimer’s risk. But should we boost the gene’s potency or suppress it? A recent discovery by Knight Initiative affiliate, Mike Greicius, may provide the answers we need.

A team of neuroscientists at the Knight Initiative for Brain Resilience have zeroed in on a critical regulator of brain metabolism that may be over-activated in the brains of patients with Alzheimer’s disease.

Groundbreaking research supported by the Knight Initiative for Brain Resilience at Stanford’s Wu Tsai Neurosciences Institute deepens our understanding of how the amino acid taurine may help reduce appetite and prevent obesity.

The Knight Initiative's Year-End Symposium and Research Showcase featured a wide range of promising new approaches to understanding brain aging, resilience, and neurodegeneration.